Why does depression cause brain atrophy in certain regions? |
- Why does depression cause brain atrophy in certain regions?
- Since insulin regulates the production of ketones, what prevents Ketoacidosis when a Type 1 Diabetic attempts a ketogenic diet?
- Is it possible to catch a second strain of the "Common Cold" while still suffering from a first?
- How does Swine Flu Work?
- Increased ocean temperature affects hurricanes (https://www.c2es.org/content/hurricanes-and-climate-change/), but does the increase in land temperature have any effect on hurricanes such as in their severity or sustainment on land?
- How exactly did pectoral fins evolve on fish?
- What happens to the suprarenal glands after a kidney removal?
- is paracetamol a nsaid?
- How does spacecraft fairing size affect the rocket handling?
- Is there a correlation between "Adrenal Fatigue" and Hypocortisolism (or Blunted Cortisol Response)?
- what’s the difference between rh-null and rh-negative?
- What happens when a person takes antibiotics?
- How do cells recognize each other?
- Do worms (I was thinking earthworms, but nematodes of any kind will work) have a distinct top and bottom?
- Does sound waves become muddled with noise if the medium it travels through is very hot?
| Why does depression cause brain atrophy in certain regions? Posted: 25 Nov 2021 01:48 AM PST |
| Posted: 25 Nov 2021 05:07 AM PST When a Type 1 Diabetic attempts to follow an extremely low carb diet they would have to take close to no insulin or else they would risk facing repeated low blood sugar levels which then would force them to eat carbohydrates to raise, making the diet no longer low carb. When insulin levels are cut too far down from a low carb diet, why doesn't ketoacidosis occur? Do Type 1 Diabetics on keto produce more ketones because of this? And if so, does that mean that they can shred excess fat at a more effective rate than someone without the disease? [link] [comments] |
| Is it possible to catch a second strain of the "Common Cold" while still suffering from a first? Posted: 24 Nov 2021 10:07 AM PST |
| Posted: 25 Nov 2021 09:43 AM PST It's thought that all Influenza originate from Birds, so why is that Swine Flu, being a strain of Influenza, primarily targets Pigs, and not Birds which should be their primary host they are adapted to? Is Swine Flu now primarily adapted to Pigs, having changed it primary host when splitting off into a new strain? How could that be possible considering it's fairly recent emergence? [link] [comments] |
| Posted: 24 Nov 2021 09:36 PM PST |
| How exactly did pectoral fins evolve on fish? Posted: 25 Nov 2021 08:30 AM PST My thinking is fins are probably modified from some type of spikes on the sides of early jawless fish, but I'm probably wrong. [link] [comments] |
| What happens to the suprarenal glands after a kidney removal? Posted: 24 Nov 2021 10:35 PM PST I'm studying biology for med school admission and I'm learning about glands. They "sit" on the top of each kidney, but what happens to them when the kidney is removed? Do they take the gland too? [link] [comments] |
| Posted: 25 Nov 2021 07:17 AM PST |
| How does spacecraft fairing size affect the rocket handling? Posted: 24 Nov 2021 09:47 AM PST Today's SpaceX DART launch featured a fairing that was larger in diameter than the main rocket body. Instinctively, this looks unstable, but obviously it is not (or at least, not in any way that can't be handled). Does the added drag, aerodynamic effects, or mass outside the width of the main rocket body cause much trouble? [link] [comments] |
| Is there a correlation between "Adrenal Fatigue" and Hypocortisolism (or Blunted Cortisol Response)? Posted: 24 Nov 2021 01:34 PM PST "Adrenal Fatigue" is a syndrome talked about in alternative medicine that is not accepted as a legitimate diagnosis in conventional medicine. On the other hand, mainstream science is beginning to discuss Hypocortisolism - I don't mean Addison's Disease - as a possible mechanism involved in the pathology of some condition that result from chronic or extreme stress such as PTSD, as well as other conditions such as Chronic Fatigue Syndrome and Fibromyalgia. It is speculated this is as a result of Hypothalamic Pituitary Axis dysregulation. When I read up on "retro" diagnoses such as Neurasthenia and Shell Shock, I start wondering if they were the same conditions with different names and all had this particular "blunted cortisol response to stress" phenomenon in common. As a layman with no background in medicine, when I read up on "Adrenal Fatigue" and Hypocortisolism, they sound very similar. And if it has been now recognized that some form of pseudo "Hypocortisolism" that isn't extreme enough to constitute as Addison's Disease may be behind a lot of chronic functional or mental conditions, why isn't more research being done to correct this form of Hypocortisolism? All I find on this subject is antidepressants can modulate the Hypothalamic Pituitary Axis to correct Hypercortisolism in depression, but nothing on how to fix a blunted cortisol response to stress. Many patients with PTSD are put on antidepressants, which to me sounds like reduces cortisol. Nicotine increases cortisol. Is it possible many people with mental disorders use tobacco to up regulate their cortisol response to stress? [link] [comments] |
| what’s the difference between rh-null and rh-negative? Posted: 24 Nov 2021 08:21 AM PST i've fallen down a rabbit hole of blood types and stuff lately. i know rh-null is extremely rare. around 50 people in the world have it if i'm correct. if rh- neg means it doesn't contain the rh antigen, then isn't that just rh-null [link] [comments] |
| What happens when a person takes antibiotics? Posted: 24 Nov 2021 08:06 AM PST Let's use a sinus infection as an example bacterial infection, as there are a lot of secondary symptoms, like sinus pressure, inflammation, fatigue, lack of appetite, etc, that are present secondary to the bacteria itself - and sinus infections are well known to the general public. Azithromycin dosage starts with 500 mg, followed by another 250 mg every 24 hours. After that first dose, it peaks in the blood at 2 hours in, and the patient can start feeling better after 3-4 hours (although azithromycin stays in the blood stream longer, up to 10 days even though the dosing regiment ends after 5 days). By what mechanism do antibiotics work this quickly? Are the antibiotics literally killing bacteria in that short time? Or is there something more going on, giving the immune system a break to stop producing t-cells or reducing inflammation, etc? Essentially, my question is why do antibiotics work so quickly (with azithromycin as an example, but really any fast working antibiotic is applicable) and how does the antibiotic have such a quick and marked effect on symptoms? [link] [comments] |
| How do cells recognize each other? Posted: 24 Nov 2021 08:51 AM PST In Immune by Philipp Dettmer, he describes your immune system as differentiating between your body and antigens with the analogy of checking if puzzle pieces fit together. In a bit more depth, what is happening when cells check to see if these protein "puzzle pieces" fit together? [link] [comments] |
| Posted: 23 Nov 2021 08:12 PM PST Like, is there a part of the worm that it would prefer to be touching the ground? Do they get disoriented if the wrong side of them is up? [link] [comments] |
| Does sound waves become muddled with noise if the medium it travels through is very hot? Posted: 24 Nov 2021 02:36 AM PST My rudimentary understanding of temperature has always been that particles "jiggle in place" more "violently" when their temperature increases. So, I wondered whether an increase in temperature in the medium a sound travels through, has an effect on the noise that medium introduces to a given acoustic signal. I imagine it would have no noticeable effect since sound is a more macro phenomenon, but it never hurts to ask :) [link] [comments] |
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