If SARS-CoV (2002) and SARS-CoV-19 (aka COVID-19) are so similar (same family of virus, genetically similar, etc.), why did SARS infect around 8,000 while COVID-19 has already reached 1,000,000? |
- If SARS-CoV (2002) and SARS-CoV-19 (aka COVID-19) are so similar (same family of virus, genetically similar, etc.), why did SARS infect around 8,000 while COVID-19 has already reached 1,000,000?
- Until the discussion about SARS-CoV-2, I had no idea you could be infected by a virus and yet have no symptoms. Is it possible that there are many other viruses I've been infected by without ever knowing?
- Why do coronaviruses seem to come from primarily bats?
- Since the CoronaVirus has a limited lifecycle outside of human body (up to 3 days on different surfaces) can the masks left unattended be reused?
- Why does water have a high specific heat?
- Viruses often (usually?) have high specificity to a certain species or small group thereof. What makes rabies so able to hop between distantly-related species?
- What do asymptomatic cases of COVID-19 mean in terms of herd immunity?
- A vaccine for the 2009 H1N1 virus was developed in 3 months (from when it was declared a pandemic). What is different about COVID-19 that makes the required time to develop a vaccine 12-14 months?
- I saw in a post the other day that in some places mosquitos are already active. Could this Covid 19 spread through mosquito bites like malaria did?
- What does "cytopathic effect not observed" mean in context of aerosolized SARS-CoV-2? (study linked in text)
- If some are using the old research of SARS and MERS as a basis to understanding COVID-19, why did they not just finish finding a cure? Surely adding that to the yearly flu vaccines would help. And what is to say that if COVID-19 just disappears, that they won't finish the cure?
- Is amount of exposure to Covid-19 affecting severity of symptoms a testable hypothesis?
- Scientists are debating the ethics of "human challenge studies" to speed up the development of a COVID-19 vaccine. How do scientists normally test the efficacy of vaccines without this type of study?
- If you contract a virus can your body develop certain antibodies that may evolve in your offspring making them better suited against similar future threats?
- Is there a way to account for incomplete and inconsistent testing for COVID-19?
- Does a single virus strain consist of an assortment of different viroids i.e. different function, or are all viroids of a single strain the same identical units?
- When will a vaccine be available for SARS-COV-2?
- Why are we not hearing much (if anything) about the number of unconfirmed cases of Covid19? I know we can't know it properly, but it's not even discussed and there are no attempts at even a guestimate.
- Why has the Corna-virus different names?
- What evolutionary advantage does a virus have if it kills the host?
- How is vaccine effectiveness evaluated?
- How are the projected death numbers for the COVID-19 pandemic calculated?
- Why don't we mass produce monoclonal antibodies specific to SARS-CoV-2 to be used as therapy?
- How much of one's body weight is being lifted during a push up?
| Posted: 02 Apr 2020 02:57 PM PDT So, they're both from the same family, and are similar enough that early cases of COVID-19 were assumed to be SARS-CoV instead. Why, then, despite huge criticisms in the way China handled it, SARS-CoV was limited to around 8,000 cases while COVID-19 has reached 1 million cases and shows no sign of stopping? Is it the virus itself, the way it has been dealt with, a combination of the two, or something else entirely? EDIT! I'm an idiot. I meant SARS-CoV-2, not SARS-CoV-19. Don't worry, there haven't been 17 of the things that have slipped by unnoticed. [link] [comments]  |
| Posted: 03 Apr 2020 04:04 AM PDT |
| Why do coronaviruses seem to come from primarily bats? Posted: 03 Apr 2020 07:21 AM PDT I was doing some reading and noticed that both SARS-CoV, SARS-CoV-2, and MERS were all presumed to have originated from bats. Why do viruses of this zoonotic origin seem to be on these small mammals as opposed to others and why is that so significant? [link] [comments]  |
| Posted: 03 Apr 2020 09:38 AM PDT If not, why? What are safe ways to disinfect face masks in household conditions? [link] [comments]  |
| Why does water have a high specific heat? Posted: 03 Apr 2020 08:50 AM PDT |
| Posted: 02 Apr 2020 09:04 AM PDT It's typically a big deal when a virus leaps from one species to another (e.g., humans) because viruses have to interact with proteins on cell surfaces, which differ greatly between species. Rabies, however, is very commonly transmitted from nonhumans to humans; 99% of rabies cases in the world are caused by dog bites, with the rest being mostly bats. Moreover, rabies can infect all warm-blooded animals and many cold-blooded ones. Most animals can be infected, though many never show symptoms. So, what makes rabies-causing viruses able to infect such a wide range of species? (I know that there are lots of different viruses that cause rabies, but to me this just begs the question—what makes so many viruses seemingly more capable of crossing the interspecies barrier?) [link] [comments]  |
| What do asymptomatic cases of COVID-19 mean in terms of herd immunity? Posted: 03 Apr 2020 08:57 AM PDT If some research is true and roughly 25% or more of cases are asymptomatic, are these individuals producing antibodies to defeat the virus and does this mean we might be closer to herd immunity without realizing? [link] [comments]  |
| Posted: 02 Apr 2020 11:59 PM PDT |
| Posted: 03 Apr 2020 07:16 AM PDT |
| Posted: 03 Apr 2020 06:56 AM PDT https://www.medrxiv.org/content/10.1101/2020.03.23.20039446v2.full.pdf+html "Air samples that were positive for viral RNA by RT-PCR were examined for viral propagation in 15 Vero E6 cells. Cytopathic effect was not observed in any sample, to date, and immunofluorescence and western blot analysis have not, so far, indicated the presence of viral antigens suggesting viral replication. However, the low concentrations of virus recovered from these samples makes finding infectious virus in these samples difficult. Further experiments are ongoing to determine viral activity in these samples." Does this mean the aerosolized virus is not spreading like airborne virus? Thanks [link] [comments]  |
| Posted: 03 Apr 2020 01:56 AM PDT |
| Is amount of exposure to Covid-19 affecting severity of symptoms a testable hypothesis? Posted: 03 Apr 2020 06:15 AM PDT Given a potentially high fatality rate among health care workers, it has at least been considered that the amount of viral particles taken in could potentially affect how severe the symptoms of Covid-19 will be when it comes to an average person: https://www.cebm.net/covid-19/sars-cov-2-viral-load-and-the-severity-of-covid-19/ Aside from studying cases as they happen, are there other ways of testing this hypothesis, such as laboratory tests? Are there any teams currently focused on researching this? If it were eventually confirmed, would this change the strategies we're currently using to eventually be able to cope with the disease? [link] [comments]  |
| Posted: 03 Apr 2020 09:45 AM PDT A human challenge study is where volunteers are injected with a new vaccine, then deliberately infected with the pathogen that the vaccine is meant to protect against. How can scientists determine whether a vaccine is effective if they never expose human test subjects to the pathogen it's meant to protect them from? Do they use animal models? Do they measure the antibodies in patients who have recovered from the illness and then compare them to antibodies from people who have had the vaccine? Some combination of those methods? I tried googling it, but I just got a bunch of articles about how scientists test vaccines for safety and possible side effects. Couldn't find much information about how they test for efficacy. [link] [comments]  |
| Posted: 03 Apr 2020 05:38 AM PDT |
| Is there a way to account for incomplete and inconsistent testing for COVID-19? Posted: 03 Apr 2020 08:37 AM PDT I see spikes or lulls in cases - and have no idea if these are related to testing rather than spread/control. Is there any method to adjust for this? Many weren't tested, despite symptoms, unless they had a direct link to someone with COVID-19 or had traveled. Now many still aren't tested here unless they are hospitalized, while others are tested based on exposure and not symptoms. The data I browse is https://www.worldometers.info/coronavirus/country/us/ [link] [comments]  |
| Posted: 03 Apr 2020 08:36 AM PDT Think the title explains question sufficiently, but is every single viroid of a virus the same, or are they differing in function in some ways? [link] [comments]  |
| When will a vaccine be available for SARS-COV-2? Posted: 03 Apr 2020 08:36 AM PDT I have read that clinical trials are underway for some sort of vaccine. I know there is a lot of uncertainty with any kind of answer, but a ballpark is better than nothing and a lot of us are left wondering and waiting not knowing what is going on with that. So I think on behalf of many of us I would like to hear an expert's opinion. How soon might we expect a vaccine to be ready for the first users? How long after that might it be widely available? [link] [comments]  |
| Posted: 03 Apr 2020 07:15 AM PDT How do we know whether it's much more prevalent than we think, and therefore consequently, less severe for a bigger majority of people? [link] [comments]  |
| Why has the Corna-virus different names? Posted: 03 Apr 2020 07:01 AM PDT In the media they switch around with Sars-cov2 , Covid-19 and Coronavirus. So my question is are they the same or not and if not why have they different names? [link] [comments]  |
| What evolutionary advantage does a virus have if it kills the host? Posted: 02 Apr 2020 12:01 PM PDT |
| How is vaccine effectiveness evaluated? Posted: 02 Apr 2020 10:44 PM PDT In addition to wanting to know this, I was curious if there's any metric that attempts to compare these two populations:
[link] [comments]  |
| How are the projected death numbers for the COVID-19 pandemic calculated? Posted: 02 Apr 2020 09:58 PM PDT I have searched a lot for this but couldn't find any explanation. The whitehouse says the projected deaths from the pandemic will be between 100-250K. However the current deaths are 6K with 250K infected, which means the cases are literally going to be a multiple of 16-41. Projecting using this we are looking at 1.6-4.1 million infections. This is something we have not seen before in the world. How are these numbers computed? Is there a specific field of science that studies this? [link] [comments]  |
| Why don't we mass produce monoclonal antibodies specific to SARS-CoV-2 to be used as therapy? Posted: 02 Apr 2020 06:48 PM PDT Vaccine development obviously will take a while and the brunt of the damage done by SARS-CoV-2 might already be done by the time a vaccine is ready. Meanwhile it seems like monoclonal antibodies already exist for this bug, so wouldn't it be more efficient to push more resources toward testing and upscaling the production of an antibody? Why is this not a more prominent option for therapy? [link] [comments]  |
| How much of one's body weight is being lifted during a push up? Posted: 02 Apr 2020 10:52 AM PDT |
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